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4a,c), as well as changes in specific lipid species that were influenced by EtOH treatment (Fig
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[ Recent studies have indicated that UA acts as an endogenous danger signal and at the same time triggers NOD-like receptor protein 3-dependent inflammation
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doi: 10.5534/wjmh.210063 87 EiniFKutenaeiMAZareeiFDastjerdiZSShirzeyliMHSalehiE
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